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It reduced MDA levels and nitrite accumulation (as an indicator of NO production), improved GSH content and SOD and catalase activity, prevented caspase-3 activation, restored serum corticosterone levels, and reduced the secretion of IL-6 and TNF-α

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The use of Aβ42H13L (TABLE 1), in which one of the adjacent His residues was replaced by Leu, a substitution that would preclude any bidentate Cu 2+ ligand binding, led to results consistent with the notion that bidentate Cu 2+ ligand binding of these His residues was not essential for the oxidative damage and cellular toxicity of the native peptide (115)