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cellular glutathione transport Controlling entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease NOVA Labs
NOVA Labs
Under basal conditions, DJ-1 present in the nucleus interacts with Daxx preventing the translocation of this death protein to the cytoplasm and its interaction with ASK1 following insults including oxidative stress
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In these models, SE triggers oxidative stress and excessive production of reactive oxygen species (ROS), both of which play a crucial role in the development of acquired epilepsy
cellular glutathione transport Controlling entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease NOVA Labs
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