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Due to the limited efficacy of current treatments for these conditions, neuroinflammation has emerged as a promising therapeutic target in drug discovery [28,29]
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However, elevated NKG2D ligand expressions as well as increased susceptibility to NK cells were found to be probably induced by SFN- or IR-mediated ROS elevation in both breast and lung cancer cells [234], demonstrating the complex interaction between ROS and NK cell functioning
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